KATANOSAKA Kimiaki

Profile

Title	Associate Professor
Belong to	Dept. of Biomedical Sciences Center for Clinical Examination Practicum Support Biomedical Sciences (graduate school)
Graduated	Osaka University, Graduate School of Science
Degree	Dr. of Science, Osaka University
Academic Institutional Membership	Physiological Society of Japan The Japanese Society for Comparative physiology and Biochemistry The Japan Neuroscience Society Society for Neuroscience
Field of Study	Neurophysiology, Sensory physiology
Research, Studies	Sensory transduction mechanisms of the primary sensory neurons, relating pain and thermosensation.
Curriculum Specialized	Physiology

Books Published, Translation

Guyton and Hall Textbook of Medical Physiology, 13e (Guyton Physiology) , a Japanese translation version, Ch.7 Excitation of Skeletal Muscle:Neuromuscular Transmission and Excitation-Contraction Coupling , pp.82-89 , (2018).

Academic Papers, Critique

Hotta, N. et al. Insulin potentiates the response to mechanical stimuli in small dorsal root ganglion neurons and thin fibre muscle afferents in vitro. J Physiol. 597(20):5049-5062, (2019), DOI: 10.1113/JP278527

Jin, K. et al. Corneal Sensory Experience via Transient Receptor Potential Vanilloid 1 Accelerates the Maturation of Neonatal Tearing. Am J Pathol. 189(9):1699-1710, (2019), DOI: 10.1016/j.ajpath.2019.05.015.

Katanosaka, K. et al. TRPV2 is required for mechanical nociception and the stretch-evoked response of primary sensory neurons. Sci. Rep. 8(1):16782, (2018), DOI: 10.1038/s41598-018-35049-4.

Ota, H. et al. EP2 receptor plays pivotal roles in generating mechanical hyperalgesia after lengthening contractions. Scand J Med Sci Sports.; 00:1-8. (2017), DOI: 10.1111/sms.12954.

Hayashi, K. et al. Muscular mechanical hyperalgesia after lengthening contractions in rats depends on stretch velocity and range of motion. European Journal of Pain. 21(1):125-139. (2017)

Takagishi,Y., et al, Disrupted axon-glia interactions at the paranode in myelinated nerves cause axonal degeneration and neuronal cell death in the aged Caspr mutant mouse shambling. Neurobiology of Aging 43:34-46, (2016)

Taguchi, T.etal., Peripheral and spinal mechanisms of nociception in a rat reserpine-induced pain model. Pain. 156(3):415-27

Katanosaka, Y. et al., TRPV2 is critical for the maintenance of cardiac structure and function in mice. Nature Commun., 5:3932. 2014

Ota, H. et al, TRPV1 and TRPV4 play pivotal roles in delayed onset muscle soreness. PLoS ONE 8(6):e65751. 2013

Kubo, A. et al., Extracellular matrix proteoglycan plays a pivotal role in sensitization by low pH of mechanosensitive currents in nociceptive sensory neurons. The Journal of physiology. 590(Pt 13):2995-3007. (2012)

Murase, S. et al.,Bradykinin and nerve growth factor play pivotal roles in muscular mechanical hyperalgesia after exercise (delayed-onset muscle soreness).Journal of Neuroscience. 30(10):3752-61, 2010

Katanosaka, K. et al., Contribution of TRPV1 to the bradykinin-evoked nociceptive behavior and excitation of cutaneous sensory neurons., Neuroscience Research, Vol.62(3),168-75, (2008)