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ITO Morihiro


Title Professor
Belong to Dept. of Lifelong Sports and Health Sciences
School of General Education, Selective Topics Program
Division of Student Support
Dept. of Biomedical Sciences
Biomedical Sciences (graduate school)
Center for Clinical Examination Practicum Support
Student Support Center
Health Promotion Center
NPO Volunteer Center
Center for Sports and Cultural Activities
Graduated Mie University, Graduate School of Medicine
Degree Dr. of Medicine, Mie University
Academic Institutional Membership Japanese Society for Virology
The Molecular Biology Society of Japan
Japanese Society of Radiological Technology
Field of Study Microbiology, Virology, Radiology
Research, Studies Studies on the cell Fusion Induced by Viruses, Analysis of fusion regulatory protein
Curriculum Specialized Microbiology

Academic Papers, Critique

A mutant fusion (F) protein of simian virus 5 induces hemagglutinin-neuraminidase-independent syncytium formation despite the internalization of the F protein. Virology, 2006 Mar 30;347(1):11-27

Human parainfluenza virus type 4 is incapable of evading the interferon-induced antiviral effect. J Virol, 2005 Dec;79(23):14756-68

Identification of RNA-binding regions on the P and V proteins of human parainfluenza virus type 2. Med Microbiol Immunol (Berl), 2006 Mar;195(1):29-36

Identification of paramyxovirus V protein residues essential for STAT protein degradation and promotion of virus replication. J Virol, 2005 Jul;79(13):8591-601

Recombinant Sendai viruses with L1618V mutation in their L polymerase protein establish persistent infection, but not temperature sensitivity. Virology. 2004 Nov 24;329(2):289-301

Early stage of establishment of persistent Sendai virus infection: unstable dynamic phase and then selection of viruses which are tightly cell associated, temperature sensitive, and capable of establishing persistent infection. J Virol, 2004 Nov;78(21):11939-51

Characterization of Sendai virus persistently infected L929 cells and Sendai virus pi strain: recombinant Sendai viruses having Mpi protein shows lower cytotoxicity and are incapable of establishing persistent infection.

Virology, 2003 Sep 15;314(1):110-24

Up-regulated expression of a novel gene in activated human peripheral blood mononuclear cells that is a truncated paralog of the human system L-amino acid transporter 1. Int J Biochem Cell Biol, 2002 Sep;34(9):1152-63

High resistance of human parainfluenza type 2 virus protein-expressing cells to the antiviral and anti-cell proliferative activities of alpha/beta interferons: cysteine-rich V-specific domain is required for high resistance to the interferons. J Virol, 2001 Oct;75(19):9165-76

Hemagglutinin-neuraminidase-independent fusion activity of simian virus 5 fusion (F) protein: difference in conformation between fusogenic and nonfusogenic F proteins on the cell surface. J Virol, 2001 Oct;75(19):8999-9009

An amino acid in the heptad repeat 1 domain is important for the haemagglutinin-neuraminidase-independent fusing activity of simian virus 5 fusion protein. J Gen Virol, 2000 Mar;81(Pt 3):719-27

Gene expression during osteoclast-like cell formation induced by antifusion regulatory protein-1/CD98/4F2 monoclonal antibodies (MAbs): c-src is selectively induced by anti-FRP-1 MAb. Bone, 1999 Jul;25(1):17-24

Cutting edge: primary structure of the light chain of fusion regulatory protein-1/CD98/4F2 predicts a protein with multiple transmembrane domains that is almost identical to the amino acid transporter E16. J Immunol, 1999 Mar 1;162(5):2462-6

Identification of regions on the fusion protein of human parainfluenza virus type 2 which are required for haemagglutinin-neuraminidase proteins to promote cell fusion. J Gen Virol, 1998 Feb;79 ( Pt 2):279-89

Role of a single amino acid at the amino terminus of the simian virus 5 F2 subunit in syncytium formation. J Virol, 1997 Dec;71(12):9855-8

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