伊藤　守弘　ITO Morihiro

プロフィール

職名	教授
所属	生命健康科学部 スポーツ保健医療学科 人間力創成教育院特別課題教育プログラム 学生部 生命健康科学部 生命医科学科 大学院 生命健康科学研究科 生命医科学専攻 臨床検査技術教育・実習センター 学生サポートセンター 健康増進センター ボランティア・NPOセンター 体育・文化センター
最終学歴	三重大学大学院医学系研究科
学位	博士（医学）（三重大学）
所属学会・役職	日本ウイルス学会 日本分子生物学会 日本放射線技術学会
専門分野	微生物学（特にウイルス学）、放射線技術学、予防医学
研究テーマ	ウイルスによる細胞融合の研究、細胞融合制御蛋白の解析
授業科目	微生物学、微生物学実習、分子感染論、感染防御技術論

学術論文、評論

A mutant fusion (F) protein of simian virus 5 induces hemagglutinin-neuraminidase-independent syncytium formation despite the internalization of the F protein. Virology, 2006 Mar 30;347(1):11-27

Human parainfluenza virus type 4 is incapable of evading the interferon-induced antiviral effect. J Virol, 2005 Dec;79(23):14756-68

Identification of RNA-binding regions on the P and V proteins of human parainfluenza virus type 2. Med Microbiol Immunol (Berl), 2006 Mar;195(1):29-36

Identification of paramyxovirus V protein residues essential for STAT protein degradation and promotion of virus replication. J Virol, 2005 Jul;79(13):8591-601

Recombinant Sendai viruses with L1618V mutation in their L polymerase protein establish persistent infection, but not temperature sensitivity. Virology. 2004 Nov 24;329(2):289-301

Early stage of establishment of persistent Sendai virus infection: unstable dynamic phase and then selection of viruses which are tightly cell associated, temperature sensitive, and capable of establishing persistent infection. J Virol, 2004 Nov;78(21):11939-51

Characterization of Sendai virus persistently infected L929 cells and Sendai virus pi strain: recombinant Sendai viruses having Mpi protein shows lower cytotoxicity and are incapable of establishing persistent infection.

Virology, 2003 Sep 15;314(1):110-24

Up-regulated expression of a novel gene in activated human peripheral blood mononuclear cells that is a truncated paralog of the human system L-amino acid transporter 1. Int J Biochem Cell Biol, 2002 Sep;34(9):1152-63

High resistance of human parainfluenza type 2 virus protein-expressing cells to the antiviral and anti-cell proliferative activities of alpha/beta interferons: cysteine-rich V-specific domain is required for high resistance to the interferons. J Virol, 2001 Oct;75(19):9165-76

Hemagglutinin-neuraminidase-independent fusion activity of simian virus 5 fusion (F) protein: difference in conformation between fusogenic and nonfusogenic F proteins on the cell surface. J Virol, 2001 Oct;75(19):8999-9009

An amino acid in the heptad repeat 1 domain is important for the haemagglutinin-neuraminidase-independent fusing activity of simian virus 5 fusion protein. J Gen Virol, 2000 Mar;81(Pt 3):719-27

Gene expression during osteoclast-like cell formation induced by antifusion regulatory protein-1/CD98/4F2 monoclonal antibodies (MAbs): c-src is selectively induced by anti-FRP-1 MAb. Bone, 1999 Jul;25(1):17-24

Cutting edge: primary structure of the light chain of fusion regulatory protein-1/CD98/4F2 predicts a protein with multiple transmembrane domains that is almost identical to the amino acid transporter E16. J Immunol, 1999 Mar 1;162(5):2462-6

Identification of regions on the fusion protein of human parainfluenza virus type 2 which are required for haemagglutinin-neuraminidase proteins to promote cell fusion. J Gen Virol, 1998 Feb;79 ( Pt 2):279-89

Role of a single amino acid at the amino terminus of the simian virus 5 F2 subunit in syncytium formation. J Virol, 1997 Dec;71(12):9855-8